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The authors reported that AOD-9604 stimulated lipolysis in isolated adipocytes and that chronic daily treatment in obese Zucker rats produced reductions in body weight gain of more than 50% relative to untreated controls over the treatment period, without effects on insulin sensitivity or IGF-1 levels [3]

These and other modifications presumably prevent an efficient interaction with GSH as reflected by the absent GSH-dependent reduction of oxidized roGFP2 in the presence of AtGrxS15

But on the specific question of which has been more rigorously studied in humans, SS-31 clearly leads

BPC-157 and TB-500 operate through distinct molecular mechanisms