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Apart from ER stress, liver-specific overexpression of NACHT, Leucine-rich-repeat and pyrin domain-containing protein 3 (NLRP3) induced marked HSC activation and fibrosis, indicating a possible role of inflammatory cell death in inducing HSC activation (Wree et al., 2014)

Currently in Phase 3 clinical trials and not yet FDA-approved, retatrutide is studied for its ability to amplify energy expenditure beyond what single or dual agonists achieve

Our previous study revealed that cellular GSH levels differed according to cell culture density, which is known to be associated with cellular ROS production [8]