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Although estrogen treatment has been shown to predispose ovariectomized rats to development of ulcerative colitis-induced tumor development [90], most studies to-date have shown that estrogen treatment decreases colonic paracellular permeability and reduces IBD symptom severity [89, 91, 92]

In vitro differential responses of rat and human Aryl hydrocarbon receptor to two distinct ligands and to different polyphenols

Silicon (Si)-based agents, when administered orally, can continuously generate a large amount of antioxidant hydrogen in the intestinal tract

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