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Besides, IH could promote neuronal apoptosis in normal mice and diabetes mice models, the latter had higher apoptosis levels and could be modulated by high mobility group box-1 protein (HMGB1)/toll-like receptor 4 (TLR4) signal [53]

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With the discovery of a new allosteric site of KRAS (G12C), several irreversible covalently binding inhibitors of KRAS (G12C) have emerged, raising the hope of drugging KRAS

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