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Monocytes, macrophages and other inflammatory mediators of abdominal aortic aneurysm

(1984) characterized structure-activity relationships in GHRH analogs, establishing that the first 29 amino acids contain the full biological activity and that specific substitutions can confer resistance to enzymatic degradation [1]

PINK1-mediated mitophagy as a non-canonical cardioprotective pathway Beyond the classical prosurvival cascades, PTEN-induced putative kinase 1 (PINK1) has emerged as a regulator of mitochondrial quality control through its role in mitophagy

Besides the light-regulated induction and dark inactivation of GSTs, these enzymes can also participate in the signal transduction of visible and UV light