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As such, Acsl4 deletion may limit AA-induced cytokine responses by modulating AA metabolism 34 , while GPX4 controlled LPO

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In vitro studies revealed that both oligomeric and fibrillar forms of -amyloid can inhibit H 2 O 2 production at elevated concentrations of Cu 2+ , with the fibrillar form producing less H 2 O 2 than its oligomeric counterpart does (Fang et al., 2010[131])

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