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This interaction prompts M1 polarization and triggers pro-inflammatory cytokine production, exacerbating acute pancreatitis through the NFKB/NF-B pathway

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This knowledge will be necessary for any subsequent studies involving the interaction of AX with proteins, requiring the presence of an antioxidant

While we were able to reproduce the finding that NINJ1 knockdown did not affect RSL3-induced ferroptosis, we found that NINJ1 knockdown robustly protected cancer cells against ferroptosis induced by xCT inhibitors, such as erastin and SAS