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We first examined exophers produced by neurons co-expressing mCherry and GFP::IFD-1 to find that exophers can contain extruded GFP::IFD-1 foci in this background (Fig

Iron dyshomeostasis and ferroptosis in Alzheimers disease: Molecular mechanisms of cell death and novel therapeutic drugs and targets for AD

in vivo experiments further revealed that inhibiting AGBL4 delays intracranial tumor progression and prolongs the survival of tumor-bearing animals, an effect that can be partially reversed by high MMP-1 expression (31)

Peptides tend to work via an indirect path such as upregulating endogenous antioxidant pathways or enhancing mitochondrial repair