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It serves as a model substrate in studies of glutathione peroxidase and glutathione transferase enzyme systems, and has been examined in laboratory models for its role in xenobiotic and electrophile detoxification, conjugation chemistry, and the protection of protein sulfhydryl groups

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doi: 10.1038/s41380-023-02150-8 16

Here, we have utilized liver-specific overexpression of cation transport regulator homolog 1 ( Chac1 ), a recently identified intracellular GSH-degrading enzyme, to induce hepatic GSH depletion during MASLD progression