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VEGF released from RMCs under high glucose conditions supports the survival of endothelial cells and retinal neurons in the early stages of injury and limits glucose-induced retinal vascular damage ( The neuroprotective effect of RMCs and some cytokines during the development of DR deserves attention by researchers

In vitro , AST significantly improved the viability of SH-SY5Y cells exposed to paraquat by dampening oxidative stress and curbing apoptosis

The MIP contributes with a large number of binding sites for Tc absorption

it reduces inflammation and apoptosis induced on by IL1 by blocking the NF-B signaling pathway in chondrocytes [84, 85]