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Social interaction deficits parallel evidence that prenatal cannabinoid exposure disrupts sociability via altered endocannabinoid and oxytocin signaling [54, 55, 56], while broader affective and motivational abnormalities align with persistent synaptic dysregulation in corticolimbic circuits following early-life adversity [57, 58, 59]

Subtypes A1 and A3 bind to G i -coupled GPCRs thereby inactivating adenylate cyclase and decreasing intracellular cAMP levels

The downfall of Retinyl Palmitate was due to released reports about the ingredient being correlated to sun damage and skin tumors

Life Sciences ence 55 :163-167