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Statins were initially developed to treat patients with hypercholesterolemia and its function at the molecular level is to inhibit 3-hydroxy-3-metil-glutaryl-CoA (HMG-CoA) reductase, the key regulatory enzyme of the endogenous mevalonate pathway of cholesterol synthesis (Figure 1).82,83 In this sense, HCV infected patients are at risk of cardiovascular disease and statins utilization are currently part of several guidelines to prevent cardiovascular events.82 Moreover, correlations between statins and HCV life cycle has been reported

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