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This usually leads to intestinal leakage as well as the infiltration and metastasis of toxins and pathogenic bacteria in the intestinal cavity, which will eventually cause inflammation in the whole body

Hormonal therapeutic agents may include, for example, hormone agonists, hormone antagonists (e.g., flutamide, bicalutamide, tamoxifen, raloxifene, leuprolide acetate, LUPRON, LH-RH antagonists), inhibitors of hormone biosynthesis and processing, and steroids (e.g., dexamethasone, retinoids, retinoid (deltoid), betamethasone (betamethasone), cortisol, cortisone (cortisone), prednisone, dehydrotestosterone (desotetasterone), glucocorticoids, mineralocorticoids, estrogens, testosterone, gestagen), vitamin a derivatives (e.g., total retinoic acid (ATRA))

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To understand the dynamics of this interplay in T1DM, one must go back to the relation between portal insulin and GHR expression on hepatocytes, which was so cleverly addressed by Leung et al