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Defective hepatic autophagy in obesity promotes er stress and causes insulin resistance

These findings suggest that, when evaluating the effects of amino acid-related metabolic axes, such as Gln-KG, on macrophage polarization, the key is not to simply classify them as pro-inflammatory or anti-inflammatory, but rather to focus on their integrated effects on anaplerotic flux, the spectrum of signaling metabolites, and the supply of epigenetic substrates, thereby reshaping inflammatory thresholds and reparative capacity across different timescales ( Nutrient-sensing pathways provide important clues for explaining the discrepant findings among different studies

Oxidative stress and nitration in neurodegeneration: cause, effect, or association

This observation raises some questions, i.e., does suppressing specific functions of the mitochondria associate with improvement in insulin sensitivity