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Demyelinating lesions or plaques of different sizes and locations are spread throughout the CNS, and the onset of symptoms and response to treatment is unique to each patient [2]

Chronic hypoxia for 7 or 14 d, but not 1 or 3 d, resulted in alpha-synuclein hyperphosphorylation at serine129 (-Syn p-S129) and protein aggregation, hippocampal neurodegeneration, and cognitive deficits, whereas the latter could be prevented by alpha-synuclein knockdown or an administered short peptide competing at -Syn S129

Assimilatory nitrate reduction genes ( nirA, narB ) were consistently expressed across samples at low levels (15 tpm) but did not exhibit differential regulation

Its interesting to note that hyperhomocysteinemia is linked to the onset of thrombosis, ischemic heart disease, and atherosclerosis (Kravchuk 2012