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80 Fernandez-VizarraE.BugianiM.GoffriniP.CarraraF.FarinaL.ProcopioE.et al (2007)

Increased oxidative stress is also a result of carboplatin activity on normal cells which is attributed to the bone marrow suppression and ototoxicity [118]

Epigenome-wide association studies (EWASs) have revealed stress-induced DNA methylation changes, such as hypermethylation of the NR3C1 (glucocorticoid receptor) gene in the central amygdala, which modulates visceral pain sensitivity in the gutbrain axis