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Our predictions indicated that the tags did not affect the protein structure, given the flexibility of the C-terminal regions

Studies in both normal healing and delayed healing models have shown improvements in callus volume, mineralization, bone mineral content, rate of successful union, and mechanical strength at fracture sites

These distinct mechanisms may include systemic dysfunction-induced neurologic injury, renin-angiotensin system (RAS) dysfunction, immune dysfunction, and direct viral invasion of the nervous system, as elaborated and summarized in Table 1 [55,56,57,58,59,60,61,62,63,64,65,66,67,68,69,70,71] and Fig

Nishimatsu, S