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Because the strategy lacks platelet-specific functions such as clot retraction and amplification mechanisms driven by platelet secretion, their ability to sustain long-term haemostasis is inherently incomplete, as reflected by occasional rebleeding in the tail-laceration assay (Supplementary Fig

For example, ferroptosis and macrophages are respectively found participating in heart diseases, like ischemia/reperfusion, heart failure, drug induced cardiotoxicity and so on, but few research focus on the interaction between ferroptosis and heart macrophages [119,120,121,122,123,124]

Resistance modulation action, time-kill kinetics assay, and inhibition of biofilm formation effects of plumbagin from Plumbago zeylanica Linn