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The correlation of EAAT expression with disease severity, in combination with the increased splice variant expression that can facilitate in reduced glutamate reuptake, suggests increased glutamate levels in vulnerable brain regions in Alzheimers disease

Schielke et al., 2010), which, as mentioned above, was implicated in diverse functions including adherence to epithelial cells, intracellular survival, vacuolar escape, interaction with dynein, and modulation of apoptosis/pyroptosis (Schmitt et al., 2007