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In PD models, the use of dependent sodium channel blockers reduces glutamatergic hyperactivity and inhibits abnormal glutamate release, suggesting that amino acid metabolism disturbances may contribute to neurotoxicity and oxidative toxicity, ultimately resulting in dopaminergic neuron death [85]

Quintanilla-Licea R, Vargas-Villarreal J, Verde-Star MJ, Rivas-Galindo VM, Torres-Hernndez D (2020) Antiprotozoal activity against Entamoeba histolytica of flavonoids isolated from Lippia graveolens Kunth