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Indeed, when 2-DG and metformin simultaneously target glycolysis and mitochondrial complex I, T cell metabolism is normalized and lupus disease is reduced in mice, thus highlighting the importance of both glycolysis and mitochondrial metabolism in T cell function and autoimmune diseases (74, 76)

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XL413, a cell division cycle 7 kinase inhibitor enhanced the anti-fibrotic effect of pirfenidone on TGF-beta1-stimulated C3H10T1/2 cells via Smad2/4