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Additionally, the therapy modulates gene expression by activating RNA transcription and DNA synthesis through upregulation of growth factors [e.g., vascular endothelial growth factor [VEGF], transforming growth factor- [TGF-]] and signaling pathways [e.g., mitogen-activated protein kinase (MAPK), AKT]

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The Src/c-Abl pathway is a potential therapeutic target in amyotrophic lateral sclerosis

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Cluster differentiation protein 36 (CD36) also induces FFA absorption, leading to increased cardiac lipotoxicity in cardiomyocytes

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