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doi: 10.22203/eCM.v035a22

Neuroprotective Mechanisms There is increasing attention on BPC-157 for its interaction with neurological pathways

This suggests that AIEC induces chronic intestinal inflammation in susceptible hosts by promoting microbial dysbiosis

Its believed to promote musculoskeletal repair by upregulating growth factors, stimulating the formation of new blood vessels, or modulating nitric oxide synthesis, but no mechanism of action has been proven.34 TB4, however, has known mechanisms by which it speeds recovery from injury: inhibiting inflammation, preventing cell death, and (as suspected with BPC) promoting blood vessel formation.35 On top of the benefits for physical recovery, the ability to form new blood vessels points to promising implications for cardiovascular health