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Although the pathogenesis of DCM is complex, after long-term exploration, scholars generally believe that the occurrence and development of DCM is mainly related to glucose and lipid metabolism disorders, microvascular endothelial dysfunction, apoptosis, advanced glycation end product (AGE) deposition, myocardial inflammation, and oxidative stress

Blockade of GCH1/BH4 Axis activates Ferritinophagy to mitigate the resistance of colorectal Cancer to Erastin-induced Ferroptosis

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however, in todays toxic world, low levels of glutathione are no longer only reserved for the aged