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The restricted use of mucoadhesive particle systems in the clinic teaches us an important lesson for future inhaled drug development: preclinical and clinical studies should include various cell lines, tissues and disease models that represent the complexity of human lung biology (Fig

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For instance, H3K18la transcriptionally activates VCAM1, which subsequently activates the AKT-mTOR-CXCL1 axis, ultimately provoking EMT and migration of gastric cancer cells and resulting in immunosuppression and unfavorable prognosis