hypothesis glutathione persister cells Drug-tolerant cancer are vulnerable to GPX4 inhibition JCI - CD8+ T cells
JCI CD8+ T cells sustain antitumor response by mediating crosstalk between adenosine A2A receptor and glutathione GPX4 Low GPX4 drives a sustained drug tolerant persister state in TNBC by a targetable adaptive FSP1 upregulation ScienceDirect FSP1 and histone deacetylases suppress cancer persister cell ferroptosis Science Advances Ferroptosis: Opportunities and Challenges in Cancer
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