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Conversely, excessive ROS accumulation leads to DNA damage, induction of p53 and Fas ligand (FasL), and ultimately T cell apoptosis ( Figure 1 ), which can be advantageous in terms of controlling autoimmunity ( Figure 1 Given this finite balance, it is not surprising that ROS are generated by multiple pathways following TCR activation and, in parallel, eliminated by multiple antioxidant systems

A cost-effectiveness analysis of DCD kidney transplantation

Deficiency of the purinergic receptor 2X7 attenuates nonalcoholic steatohepatitis induced by high-fat diet: possible role of the NLRP3 inflammasome

GSH toxic levels cause both potent UPR and a full iron starvationlike response To search for causes of GSH toxicity, we established the mRNA profiles of HGT1 cells cultured with 50 M GSH during 5, 30 and 240 min