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It is currently understood that small molecules capable of traversing the OM either possess specific physicochemical properties that enable crossing of this barrier by passive diffusion or hijacking porins or other membrane-embedded proteins (Fig

Zeng et al., 2007) and also accelerates tumor progression induced by KSHV-encoded oncoproteins such as G protein-coupled receptor and kaposin A

The MNs developed by the team are manufactured in a differential dual-release manner, with the outer layer replacing the aluminum adjuvant to form an antigen reservoir for sustained antigen release and the inner layer rapidly releasing the adjuvant to activate DCs, effectively reducing the amount of aluminum while maintaining its effect (Fig