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On the basis of our findings in those patients and on published experience, we concluded that anaphylactoid reactions to intravenous NAC are dose-related and the antihistamines are useful in controlling and in preventing recurrence of anaphylactoid symptoms

demonstrated that polymorphism of the optic atrophy 1 (OPA1) gene, which encodes a dynamin-related mitochondrial protein, was related to the pathology of OAG.[43] Additionally, we previously showed that mitochondrial DNA damage in blood cells, represented by an increased mitochondrial/nuclear DNA (mtDNA/nDNA) ratio, was associated with abnormal BF (MBR) in the ONH in glaucoma patients.[44] Taking these past results together with those of this study, it is thus reasonable to consider that systemic mitochondrial dysfunction influences hemodynamics in the ONH