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Collectively, these observations highlight a convergent pathogenic mechanism in NDDs, in which mitochondrial quality control failure, through impaired mitophagy, defective protein import, and ROS-driven dysfunction, links mitochondrial damage, mtDNA-mediated inflammation, and protein aggregation [68,69,70,71,72,73]

Implication of leukocytes in the oxidative stress of senescence

Apoptotic cells typically upregulate the glycolytic pathway to provide additional energy, and mitochondria also participate in regulating apoptosis

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