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Ferroptosis and AD pathology Amyloid (A) plaques and neurofibrillary tangles (NFTs) A plaque accumulation is a hallmark pathological feature of AD, where abnormal A aggregation disrupts synaptic function and impairs memory (30,31)

Another study reported significantly elevated agmatine concentrations in both the plasma and postmortem frontal cortex tissue of first-episode and chronic schizophrenia patients (9093), and found that antipsychotic treatment could reduce blood agmatine levels (94)

Saturated fatty acids liberated from LDs, or taken up from circulation into the liver, may directly promote tissue injury, leading to additional macrophage recruitment and activation (121)