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Conversely, the conversion of Cu(II) to the more toxic Cu(I) induces cell death and intracellular oxidative stress, leading to an increase in heat shock protein 70 expression levels (190)

Dayne Curtis Thomas Jr., senior, Deans List, College of Arts and Sciences, biological sciences

10.3389/fonc.2022.833814 41 LiL

91 showed that PFD delayed OA progression by inhibiting the proliferation and migration of FLSs stimulated by TGF-1, while decreasing the expression Col1a1 , TIMP-1 , and ACTA-2 (-SMA), and IL-6 without affecting TNF- levels, thereby alleviating synovial inflammation and fibrosis, as evidenced by improved OARSI scores in OA models