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Prior results from our lab have shown that the role of Cn Acs1 in carbon metabolism is even more pronounced during infection 23 than under in vitro conditions mimicking the host environment, supporting the possibility that the efficacy of Cn Acs1 inhibitors is likely to be increased in vivo relative to in vitro

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Thiamine deficiency in diabetes mellitus and the impact of thiamine replacement on glucose metabolism and vascular disease

The 5-FU mouse model was selected because it is rapidly developed, cost-effective, and closely reflects the pathological features observed in human CIM, making it a widely used approach for evaluating therapeutic candidates (Huang et al