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suggest that the pathogenesis of mitochondrial-associated epilepsy involves processes such as neuronal energy depletion, oxidative stress, impaired calcium signaling, neurotransmitter imbalances, -oxidation deficiency, and glial cell dysfunction (3)

It relies on our movements to keep it flowing

Spatiotemporal profiling defines persistence and resistance dynamics during targeted treatment of melanoma

A reduction in intracellular lactate levels, primarily due to excessive efflux and consumption by cardiomyocytes, contributes significantly to diminished lactylation at the K1897 site of -myosin heavy chain during myocardial injury [62]