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When blood vessels first constrict and then dilate, the resulting surge of blood flow could theoretically deliver more peptide to target areas

Sequencing interventions strategically maximizes this force multiplier effect

Once intracellular, Mtb itself attempts to evade the macrophages killing processes by secreting virulence factors that inhibit the alveolar macrophages expression of the nuclear factor-B (NF-B) and IFN, ultimately promoting Mtb s intracellular survival (151, 157)

Importantly, this analysis indicated that the majority of targeted genes are not generally required for intestinal gene expression or GFP stability