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Critical depletion of GSH in mitochondria culminates in increased levels of ROS and RNS, mitochondrial dysfunction and subsequent ATP depletion, which are responsible for the shift from apoptosis towards necrosis and, therefore, inflammation

Whether these tissues (other than the liver) were capable of metabolizing acetone or whether the metabolites themselves were distributed to the tissues was unclear

m - o , Levels of various nutrients in the administered diet (yellow) and in Het (blue) or KO (red) stool from microbiota-depleted mice fed on a No Cys diet for 3 days