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Mitophagy serves as a protective mechanism against excessive mtROS by selectively degrading damaged mitochondria, as observed with IL-10 antagonism of LPS triggered inflammation 30 , while the absence of autophagy also results in ROS-dependent amplification of retinoic acid-inducible gene I (RIG-I)-like signalling 71,72,73

Golgi staining of aged astrocytes did not show major morphological changes 529 , whereas age-dependent changes in astrocytes stained with antibodies against S100B and glutamine synthetase are characterised by complex region-dependent changes ranging from atrophy to hypertrophy

De la Pena JA, Rubio OV, Cano JP et al