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Interestingly, the expressions of hsa-mir-122-5p, IL6, FGL1, ACADM, LEP, INS, CASP1, NLRP3, ACAT1, TNF, ELAVL1, IGFBP7, ADAM9, GDF15, ACADVL, ECH1, octanoylcarnitine (octanoylcarnit.), isoleucine, leucine, L-valine, 7-keto-8-aminopelargonic acid (7-keto-8-amino.), creatinine, decanoylcarnitine (decanoylcarnit.), and hexanoylcarnitine (hexanoylcarnit.) were found to be upregulated in DCM, while those of hsa-mir-30c-5p, hsa-mir-30d-5p, hsa-mir-22-3p, GSTM2, GPX3, GSTM3, ADIPOQ, HADH, PRDX2, PRDX1, SERPINA1, A2M, PRDX6, APOA1, APCS, NPPA, bilirubin, butyric acid (butyrate), alanine, glutamine, and cytidine triphosphate (ara-CTP) were found to be downregulated in DCM

Bhatia 2014

Additionally, it is also crucial to evaluate whether free covalent inhibitors can be eliminated by metabolic enzymes in vivo during the development of covalent inhibitors

The primary pathological hallmark of PD is the presence of Lewy bodies, which are intracellular aggregates of the protein alpha-synuclein