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Oxidatively truncated phospholipids are required agents of tumor necrosis factor alpha (TNFalpha)-induced apoptosis

These results corroborate our previous findings ( Ferroptosis, a form of iron-dependent cell death characterized by oxidative lipid damage and impaired antioxidant capacity, has emerged as a key player in cisplatin nephrotoxicity ( , specifically its subunit xCT, impairs cystine uptake and GSH synthesis, weakening the antioxidant capacity of GPX4, the primary enzyme responsible for reducing lipid hydroperoxides

During a median follow-up of 6.8 years after the on-treatment LSM, 53 patients developed HCC

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