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6ag, and Supplementary Fig

The public COA library was a deciding factor no other supplier offers that level of transparency. We source BPC-157 from several suppliers for comparative studies

When lysosomal function is severely impaired, lysosome-independent degradation pathways, such as those activated by Bortezomib, a proteasome modulator, can enhance ubiquitination and proteasomal activity, compensating for defective lysosomal function and facilitating the removal of damaged proteins and organelles, thus improving immunotherapy efficacy (133) ( Table 1 )