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doi: 10.1007/s13402-024-00920-2 57 LiuCXGaoYXuXFJinXZhangYXuQet al

Pearson-Smith & Patel, 2017

Previous studies in STZ-type 1 diabetic rats revealed that ESC also reverses the global loss of H3K9/14 acetylation and H3K4 monomethylation, including at Mmp13/Bmp6 sites, leading to normalization of profibrotic signaling and glomerulosclerosis (Surse et al