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Overexpression of TFEB leads to degradation of bulk autophagic substrates such as damaged mitochondria (Nezich et al., 2015), long-lived protein aggregates (Martini-Stoica et al., 2016), and lipid droplets (Settembre et al., 2013), indicating the crucial role of TFEB in organelle-specific autophagy, such as mitophagy and lipophagy (Settembre et al., 2013

The conversion of bacteria to L-forms in adverse environments is also a potential cause of antibiotic resistance [16, 20, 27]

On the other hand, an increase in the expression of this protein was observed in HT-22 co-treated with H

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