glutathione pathway to triple negative cancer NRF2 activation by cysteine as a survival mechanism for triple-negative breast cells Metabolic interplay between exogenous cystine
Metabolic interplay between exogenous cystine and glutamine dependence in triple negative breast cancer Cell Death Discovery Activation of the eIF2 ATF4 axis drives triple negative breast cancer radioresistance by promoting glutathione biosynthesis ScienceDirect Adipocyte derived glutathione promotes obesity related breast cancer by regulating the SCARB2 ARF1 mTORC1 complex: Cell Metabolism International Journal of Oncology
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