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As our results show that SNIP1 and CDK11 act together in spliceosome activation and CDK11 is overexpressed in many cancers 8,104,105,106,107,108 , the CDK11/P-SF3B1/SNIP1 pathway may be a novel oncogenic driver of splicing-dependent cancers 109 , warranting further investigation

On the other hand, cells incubated with AuNPs-d-PLL-FA.siRNA (25 nM) presented a mean percent fluorescence of ~ 17%, indicating some interaction with PC-3 PSMA cells