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[DOI] [PubMed] [Google Scholar] 77.Wang J, Lee S, Teh CE-Y, Bunting K, Ma L, Shannon MF

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The failure of macrophages to clear apoptotic cells contributes to prolonged inflammation, as these unengulfed cells can undergo secondary necrosis, releasing inflammatory mediators that exacerbate tissue damage and promote a new cycle of inflammation (134, 135)

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demonstrated that diabetic rats with ulcerated skin show a deficiency in GSH synthetase in granulation tissue keratinocytes, resulting in reduced GPX4 activity 42

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