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Loss of the neurotransmitter dopamine, due to the degeneration of dopaminergic neurons in the midbrain underlies the pathophysiology of the motor dysfunction

Fifth, S-adenosylmethionine (SAM), the methyl-donating substrate of histone methyltransferases, and S-adenosylhomocysteine (SAH) link one-carbon metabolism to methylation status

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Impairments in neuronal survival mediated by HMGB1 are believed to be closely associated with the glycolytic pathway, as evidenced by the mitigation of these adverse effects through treatment with the glycolysis inhibitor 2-DG [194]