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In addition, as PAH mediates the conversion of phenylalanine (Phe) to tyrosine (Tyr), hyperphenylalaninemia (HPA) is present in all BH 4 deficiencies apart from autosomal dominant guanosine triphosphate cyclohydrolase I deficiency (AD-GTPCHD) and sepiapterin reductase deficiency (SRD) [1, 2]

The KPV fragment retains the anti-inflammatory properties of alpha-MSH but lacks the receptor-binding activity that causes melanocortin side effects such as skin darkening or appetite changes

Similarly, treatment of NSD2 HiBit cells with 1 M UNC10415667 for 6 h resulted in a reduction of NSD2 levels by about 75% (Fig

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