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From the previous section, it follows that inherited disorders leading to dramatic elevations of homocysteine in the blood and urine derive from genetic defects in homocysteine transsulfuration or re-methylation, or in the transporters and enzymes involved in the delivery and metabolism of the key cofactors required for these metabolic pathways (specifically folate and vitamin B12)

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Indeed, SLC1A5 is highly expressed in various solid cancers 73,76,77 , such as squamous lung cancer, in which it is responsible for approximately 50% of glutamine uptake 73