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Once senescence is established, the cell enters a state of permanent arrest and exhibits specific markers, such as -galactosidase activity, elevated levels of p53 and p16INK4a, and changes in the chromatin landscape that reinforce its non-proliferative state.1 Understanding the molecular mechanisms underlying senescence is critical for developing interventions in aging-related diseases and cancer

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Second, the development of sophisticated delivery systems, such as nanoparticles and biomimetic vesicles, is crucial to enhance the bioavailability and tumor-specific targeting of ferroptosis inducers while minimizing off-target toxicity to normal tissues