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This cell-to-cell adhesion is translated into intracellular signaling pathways [37], culminating in specific alterations within the cell, at the cytoplasmatic level

Insulin-dependent activation of endothelial nitric oxide synthase is impaired by O-linked glycosylation modification of signaling proteins in human coronary endothelial cells

2.2 Immune pathway crosstalk Th17 cells, the interleukin (IL)-17 producing CD4+ T helper cells, have gained significant attention due to their crucial role as important mediators of immune dysregulation, for their production of signature cytokines including IL17 and IL-17F under the induction by pathogens

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